Author

Derin I. Yetil

New York Institute of Technology

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Ashwin RagupathiiD· 2 shared Binghui SheniD· 2 shared Dong ZhangiD· 2 shared Heba Zuhair AbidiD· 2 shared Huaiying ZhangiD· 2 shared Ming XiaoiD· 2 shared Rongwei ZhaoiD· 2 shared Anthony FernandeziD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersMolecular Biology

Ashwin RagupathiORCID

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2 papers

Recent·Most cited

The function of human PIF1 in G quadruplex formation and replication stress response at ALT telomeres

Ashwin Ragupathi, Rongwei Zhao, Shakir Najit, Anthony Fernandez, Heba Zuhair Abid, Jiho Mo, Samantha Juang, Christopher Tong, Derin I. Yetil, Liam Buchanan, Connor McDowell, Lindsay Mosca, Bronwyn Kelly, Rebecca Kelly, Zakir Hossain, , Dong Zhang

This study investigates the role of human PIF1 in ALT telomere maintenance, showing its involvement in DNA damage, G4 formation, and replication stress response, with implications for cancer therapy.

bioRxiv · Sep 1, 2026 · PreprintMolecular Biology

DNA2 and FANCM function in two distinctive pathways in disrupting TERRA R-loops and suppressing replication stress at ALT telomeres

Ashwin Ragupathi, Heba Zuhair Abid, Yun Chen, Rongwei Zhao, Settapong T Kosiyatrakul, Derin I. Yetil, Julia Neiswander, Rachel Feltman, Shannon Thomas, Benjamin Yusupov, Manrose Singh, Zheng Li, Binghui Shen, Huaiying Zhang, Hsueh-Ping C. Chu, , Dong Zhang

This study shows DNA2 and FANCM function in distinct pathways to suppress replication stress at ALT telomeres, with their combined depletion causing synthetic lethality in ALT+ cancers.

bioRxiv · May 22, 2025 · PreprintCancer Biology