Author

E. Clayer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Bankovacki· 1 shared A. I. Webb· 1 shared A. J. Kueh· 1 shared C. Stafford· 1 shared D. Dalseno· 1 shared D. Lacey· 1 shared E. Carr· 1 shared F. M. Hansen· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. Bankovacki

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2 papers

Recent·Most cited

Severe dysregulation of TNF expression leads to multiple inflammatory diseases and embryonic death

P. Bouillet, E. Clayer, D. Dalseno, A. J. Kueh, D. Lacey, M. Tsai, E. Carr, V. Wimmer

The paper identifies a new regulatory element in the Tnf 3UTR that cooperates with existing elements to downregulate TNF expression and prevent autoimmune diseases, and shows excessive TNF causes embryonic death.

bioRxiv · May 28, 2020 · PreprintImmunology

A regulatory interface on RIPK2 is required for XIAP binding and NOD signaling activity

V. J. Heim, L. F. Dagley, C. Stafford, F. M. Hansen, E. Clayer, A. Bankovacki, A. I. Webb, I. S. Lucet, J. Silke, U. Nachbur

The paper identifies a regulatory interface on RIPK2 essential for XIAP binding and NOD signaling by analyzing post-translational modifications in RIPK2 knock-in mice.

bioRxiv · Mar 12, 2020Cell Biology