Author

E. Miranda

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Cagan· 1 shared A. Carrasco· 1 shared A. M. Flanagan· 1 shared A. Melnick· 1 shared A. Vilas-Zornoza· 1 shared B. Paiva· 1 shared C. C. Brown· 1 shared C. Meydan· 1 shared

Activity by Year

2018-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

A. Cagan

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Chromatin activation as a unifying principle underlying pathogenic mechanisms in multiple myeloma

R. Ordonez, M. Kulis, N. Russinol, V. Chapaprieta, R. Beekman, C. Meydan, M. Duran-Ferrer, N. Verdaguer-Dot, G. Clot, R. Vilarrasa-Blasi, L. Garate, E. Miranda, A. Carrasco, T. Ezponda, A. Vilas-Zornoza, , J. I. Martin-Subero

The paper identifies aberrant chromatin activation as a common mechanism driving the malignant phenotype in multiple myeloma.

bioRxiv · Aug 20, 2019Cancer Biology

A novel innate lymphoid cell delineates childhood autoimmune arthritis

M. D. C. Velasco-Herrera, M. D. Young, F. A. V. Braga, E. C. Rosser, E. Miranda, L. Marshall, M. G. L. Wilkinson, L. Mamanova, M. Efremova, R. Vento-Tormo, A. Cagan, H. Al-Mossawi, S. Teichmann, A. M. Flanagan, L. Wedderburn, , C. C. Brown

The paper identifies a novel synovial innate lymphoid cell (ILC) subtype associated with a specific type of childhood autoimmune arthritis, highlighting its role in inflammation and potential as a therapeutic target.

bioRxiv · Sep 20, 2018 · PreprintImmunology