Author

E. Sloan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Assay:bulk RNA-seq· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:protein folding & dynamics· 1 Organism:rat· 1 Organism:virus· 1

Frequent Co-Authors

E. Hutchinson· 3 shared C. Boutell· 2 shared A. B. Russell· 1 shared A. da Silva Filipe· 1 shared A. Mehle· 1 shared A. Sims· 1 shared C. B. Brooke· 1 shared C. Loney· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersMicrobiology

E. Hutchinson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cryptic proteins translated from deletion-containing viral genomes dramatically expand the influenza virus proteome

J. N. Ranum, M. P. Ledwith, F. G. Alnaji, M. Diefenbacher, R. Orton, E. Sloan, M. Guereca, E. M. Feltman, K. Smollett, A. da Silva Filipe, M. Conley, A. B. Russell, C. B. Brooke, E. Hutchinson, A. Mehle

The paper identifies cryptic proteins from deletion-containing viral genomes that inhibit influenza virus replication and expand its proteome.

bioRxiv · Dec 12, 2023Microbiology

Superinfection exclusion creates spatially distinct influenza virus populations

A. Sims, L. B. Tornaletti, S. Jasim, C. Perillo, R. Devlin, J. Hirst, C. Loney, J. Wojtus, E. Sloan, L. Thorley, C. Boutell, E. Roberts, E. Hutchinson

This study shows that superinfection exclusion (SIE) creates spatially distinct influenza virus populations, limiting genome interactions between virus subpopulations as they spread within host tissues.

bioRxiv · Jun 6, 2022Microbiology

Constitutive TRIM22 expression within the respiratory tract identifies tissue-specific and cell-type dependent intrinsic immune barriers to influenza A virus infection

M. Charman, S. McFarlane, J. K. Wojtus, E. Sloan, R. Dewar, G. Leeming, M. Al-Saadi, L. Hunter, M. Carroll, J. P. Stewart, P. Digard, E. Hutchinson, C. Boutell

This study identifies TRIM22 as a constitutively expressed antiviral factor in the respiratory tract that restricts influenza A virus infection independently of cytokine-mediated immunity.

bioRxiv · Jun 21, 2019 · PreprintImmunology