Author

E. Vazquez

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Assay:metagenomics· 1

Frequent Co-Authors

A. Dopazo· 2 shared A. Benguria· 1 shared A. Efeyan· 1 shared A. Hernandez-Barranco· 1 shared A. Ortega-Molina· 1 shared A. Quintas· 1 shared A. Saez· 1 shared B. Herrero-Fernandez· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersImmunology

A. Dopazo

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Role of Lamin A/C on dendritic cell function in antiviral immunity

B. Herrero-Fernandez, R. Gomez-Bris, M. Ortega-Zapero, A. Saez, S. Iborra, V. Zorita, A. Quintas, E. Vazquez, A. Dopazo, F. Sanchez-Madrid, S. M. Arribas, J. M. Gonzalez-Granado

This study shows that lamin A/C is essential for dendritic cell function in antiviral immunity by modulating epigenetic and transcriptional processes.

bioRxiv · May 14, 2024Immunology

NGFR regulates germinal center B-cell activation and negative selection

A. Hernandez-Barranco, M. S. Mazariegos, V. Santos, E. Caleiras, L. Nogues, F. Mourcin, S. Leonard, C. Oblet, S. Genebrier, D. Rossille, A. Benguria, E. Vazquez, A. Dopazo, A. Efeyan, A. Ortega-Molina, , H. Peinado

This study shows that NGFR regulates germinal center B-cell activation and negative selection by maintaining GC structure and function, affecting antibody production and immune tolerance.

bioRxiv · Jul 14, 2023 · PreprintImmunology

Expression of the cancer-associated DNA polymerase ϵ P286R in fission yeast leads to translesion synthesis polymerase dependent hypermutation and defective DNA replication

I. Soriano, E. Vazquez, N. de Leon, S. Betrand, E. Heitzer, S. Toumazou, C. Palles, C.-C. Pai, T. Humphrey, I. Tomlinson, S. Cotterill, S. Kearsey

This paper models the POLE-P286R mutation in fission yeast, showing it causes hypermutation, DNA replication defects, and dependence on translesion synthesis polymerases.

bioRxiv · Apr 6, 2021Cancer Biology