Author

F. Samaniego

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

H. Yang· 2 shared L. Nastoupil· 2 shared M. R. Green· 2 shared Q. Deng· 2 shared A. Bagaev· 1 shared A. Haouz· 1 shared A. Mechaly· 1 shared A. Wilson· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCancer Biology

H. Yang

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

CREBBP lysine acetyltransferase domain mutations create zombie enzymes that alter chromatin loading dynamics and prevent EP300 redundancy

H. Yang, W. Zhang, V. Ravanmehr, A. Mechaly, L. Regad, A. Haouz, R. Chen, J. Henderson, E. Rojas, A. Wilson, S. Parsons, L. Nastoupil, R. E. Davis, Q. Deng, F. Samaniego, , M. R. Green

The paper shows that CREBBP mutations disrupt chromatin loading dynamics and EP300 redundancy, creating zombie enzymes that impair enhancer activation in GC B-cells and lymphoma.

bioRxiv · Sep 5, 2023 · PreprintCancer Biology

Immune microenvironment subtypes and association with tumor cell mutations and antigen expression in follicular lymphoma.

G. Han, Q. Deng, E. Dai, M. Dang, J. Ma, H. Yang, O. Kudryashova, M. Meerson, S. Isaev, N. Kotlov, K. Nomie, A. Bagaev, S. Parmar, F. Hagemeister, S. Ahmed, , M. R. Green

The paper identifies immune microenvironment subtypes in follicular lymphoma linked to tumor mutations and antigen expression, informing potential immunotherapeutic strategies.

bioRxiv · Apr 27, 2021 · PreprintCancer Biology

Overcoming ibrutinib resistance by targeting phosphatidylinositol-3-kinase signaling in diffuse large B-cell lymphoma

N. Jain, O. Havranek, R. K. Singh, T. Khashab, F. Shirazi, F. Samaniego, L. Sehgal

This study identifies phosphatidylinositol-3-kinase-beta upregulation as a mechanism of ibrutinib resistance in diffuse large B-cell lymphoma and shows that targeting this pathway with KA2237 can overcome resistance.

bioRxiv · Jan 28, 2019 · PreprintCancer Biology