Author

G. E. Himmler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Research Focus:benchmarking· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

E. R. Mackow· 2 shared J. N. Conde· 2 shared M. C. Mladinich· 2 shared W. R. Schutt· 2 shared A. A. Amarilla· 1 shared A. Patel· 1 shared C. J. Hastings· 1 shared C. N. H. Marques· 1 shared

Activity by Year

2023-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersMicrobiology

E. R. Mackow

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ZIKV Induction of Tristetraprolin in Endothelial and Sertoli Cells Post-Transcriptionally Inhibits IFNβ/{lambda} Expression and Promotes ZIKV Persistence

W. R. Schutt, J. N. Conde, M. C. Mladinich, G. E. Himmler, E. R. Mackow

This study shows ZIKV induces TTP in endothelial and Sertoli cells, which post-transcriptionally inhibits IFNβ/λ secretion, promoting viral persistence.

bioRxiv · May 3, 2023Microbiology

Establishment of a CPER Reverse Genetics System for Powassan Virus Defines Attenuating NS1 Glycosylation Sites and an Infectious NS1-GFP11 Reporter Virus

J. N. Conde, G. E. Himmler, M. C. Mladinich, Y. X. Setoh, A. A. Amarilla, W. R. Schutt, N. Saladino, E. E. Gorbunova, D. J. Salamango, E. Wimmer, H. K. Kim, E. R. Mackow

This paper establishes a reverse genetics system for Powassan virus, identifies attenuating NS1 glycosylation sites, and creates an infectious NS1-GFP11 reporter virus to study viral functions.

bioRxiv · May 3, 2023Microbiology

Immune response modulation by Pseudomonas aeruginosa persister cells

C. N. H. Marques, C. J. Hastings, G. E. Himmler, A. Patel

This study shows Pseudomonas aeruginosa persister cells evade host innate immunity by resisting complement-mediated killing and inducing a shift in macrophage polarization from M2 to M1 over time.

bioRxiv · Jan 7, 2023 · PreprintMicrobiology