Author

G. Friedman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:mouse· 1

Frequent Co-Authors

M. Pevsner-Fischer· 2 shared O. Levi-Galibov· 2 shared R. Nevo· 2 shared R. Scherz-Shouval· 2 shared Y. Stein· 2 shared A. Giladi· 1 shared A. Mayo· 1 shared C. A. Iacobuzio-Donahue· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

M. Pevsner-Fischer

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

BRCA mutational status shapes the stromal microenvironment of pancreatic cancer linking CLU+ CAF expression with HSF1 signaling

L. Shaashua, M. Pevsner-Fischer, G. Friedman, O. Levi-Galibov, S. Nandakumar, R. Nevo, L. E. Brown, W. Zhang, Y. Stein, H. S. Kim, L. Bojmar, W. R. Jarnagin, N. Lecomte, R. Stok, H. Bishara, , R. Scherz-Shouval

This study shows BRCA mutations in pancreatic cancer alter CAF subtypes, specifically increasing CLU+ CAFs, and links this to HSF1 signaling.

bioRxiv · Aug 18, 2021 · PreprintCancer Biology

Cancer-associated fibroblast compositions change with breast cancer progression linking S100A4 and PDPN ratios with clinical outcome

G. Friedman, O. Levi-Galibov, E. David, C. Bornstein, A. Giladi, M. Dadiani, A. Mayo, C. Halperin, M. Pevsner-Fischer, H. Lavon-Pashot, R. Nevo, Y. Stein, H. R. Ali, C. Caldas, E. Nili-Gal-Yam, , R. Scherz-Shouval

The paper tracks changes in cancer-associated fibroblast (CAF) subpopulations during breast cancer progression, linking their ratios to clinical outcomes and BRCA mutations.

Nature Cancer · Jun 29, 2020Peer-reviewedCancer Biology