Author

O. Levi-Galibov

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 4

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 4 Research Focus:cell type annotation· 4 Organism:human· 4 Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

R. Scherz-Shouval· 4 shared R. Nevo· 3 shared Y. Stein· 3 shared G. Friedman· 2 shared M. Dadiani· 2 shared M. Pevsner-Fischer· 2 shared A. Ben-Shmuel· 1 shared A. Giladi· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

4 shared papersCancer Biology

R. Scherz-Shouval

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cancer-associated fibroblasts serve as decoys to suppress NK cell anti-cancer cytotoxicity

A. Ben-Shmuel, Y. Gruper, O. Levi-Galibov, H. Rosenberg-Fogler, G. Carradori, Y. Stein, M. Dadiani, M. Naumova, R. Nevo, D. Morzaev-Sulzbach, G. Yagel, S. Mayer, E. Gal-Yam, R. Scherz-Shouval

The paper shows cancer-associated fibroblasts suppress NK cell cytotoxicity by acting as decoys, leading to poor disease outcomes in breast cancer.

bioRxiv · Nov 23, 2023 · PreprintCancer Biology

BRCA mutational status shapes the stromal microenvironment of pancreatic cancer linking CLU+ CAF expression with HSF1 signaling

L. Shaashua, M. Pevsner-Fischer, G. Friedman, O. Levi-Galibov, S. Nandakumar, R. Nevo, L. E. Brown, W. Zhang, Y. Stein, H. S. Kim, L. Bojmar, W. R. Jarnagin, N. Lecomte, R. Stok, H. Bishara, , R. Scherz-Shouval

This study shows BRCA mutations in pancreatic cancer alter CAF subtypes, specifically increasing CLU+ CAFs, and links this to HSF1 signaling.

bioRxiv · Aug 18, 2021 · PreprintCancer Biology

Evolution of metastases-associated fibroblasts in the lung microenvironment is driven by stage-specific transcriptional plasticity

O. Shani, Y. Raz, L. Monteran, O. Megides, H. Shacham, N. Cohen, D. Silverbush, O. Levi-Galibov, C. Avivi, R. Sharan, A. Madi, R. Scherz-Shouval, I. Barshack, I. Tsarfaty, N. Erez

This study shows lung fibroblasts in breast cancer metastases are transcriptionally dynamic and plastic, with stage-specific gene signatures and Myc as a key regulator linked to poor survival.

bioRxiv · Jul 6, 2020 · PreprintCancer Biology

Cancer-associated fibroblast compositions change with breast cancer progression linking S100A4 and PDPN ratios with clinical outcome

G. Friedman, O. Levi-Galibov, E. David, C. Bornstein, A. Giladi, M. Dadiani, A. Mayo, C. Halperin, M. Pevsner-Fischer, H. Lavon-Pashot, R. Nevo, Y. Stein, H. R. Ali, C. Caldas, E. Nili-Gal-Yam, , R. Scherz-Shouval

The paper tracks changes in cancer-associated fibroblast (CAF) subpopulations during breast cancer progression, linking their ratios to clinical outcomes and BRCA mutations.

Nature Cancer · Jun 29, 2020Peer-reviewedCancer Biology