Author

M. Pevsner-Fischer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Organism:human· 3 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 1

Frequent Co-Authors

R. Scherz-Shouval· 3 shared Y. Stein· 3 shared C. Halperin· 2 shared G. Friedman· 2 shared O. Levi-Galibov· 2 shared R. Nevo· 2 shared U. Alon· 2 shared A. Giladi· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCancer Biology

R. Scherz-Shouval

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A cell circuit approach to dissect fibroblast-macrophage interactions in the tumor microenvironment

S. Mayer, T. Milo, A. Isaacson, C. Halperin, S. Miyara, Y. Stein, M. Pevsner-Fischer, E. Tzahor, U. Alon, R. Scherz-Shouval

The paper uses a cell circuit approach to analyze fibroblast-macrophage interactions in the tumor microenvironment, finding that fibroblasts rely on autocrine growth factors while macrophages depend on external cues.

bioRxiv · Nov 17, 2022 · PreprintCancer Biology

BRCA mutational status shapes the stromal microenvironment of pancreatic cancer linking CLU+ CAF expression with HSF1 signaling

L. Shaashua, M. Pevsner-Fischer, G. Friedman, O. Levi-Galibov, S. Nandakumar, R. Nevo, L. E. Brown, W. Zhang, Y. Stein, H. S. Kim, L. Bojmar, W. R. Jarnagin, N. Lecomte, R. Stok, H. Bishara, , R. Scherz-Shouval

This study shows BRCA mutations in pancreatic cancer alter CAF subtypes, specifically increasing CLU+ CAFs, and links this to HSF1 signaling.

bioRxiv · Aug 18, 2021 · PreprintCancer Biology

Cancer-associated fibroblast compositions change with breast cancer progression linking S100A4 and PDPN ratios with clinical outcome

G. Friedman, O. Levi-Galibov, E. David, C. Bornstein, A. Giladi, M. Dadiani, A. Mayo, C. Halperin, M. Pevsner-Fischer, H. Lavon-Pashot, R. Nevo, Y. Stein, H. R. Ali, C. Caldas, E. Nili-Gal-Yam, , R. Scherz-Shouval

The paper tracks changes in cancer-associated fibroblast (CAF) subpopulations during breast cancer progression, linking their ratios to clinical outcomes and BRCA mutations.

Nature Cancer · Jun 29, 2020Peer-reviewedCancer Biology