Author

G. Marteil

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. S. Ribeiro· 1 shared B. P. de Almeida· 1 shared B. Sousa· 1 shared C. Horta· 1 shared I. S. Fonseca· 1 shared J. B. P. Leal· 1 shared J. C. Vaz· 1 shared J. P. Taborda Barata· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

A. S. Ribeiro

Mutual co-authors in cluster

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2 papers

Recent·Most cited

High prevalence and dependence of centrosome clustering in mesenchymal tumors and leukemia

N. M. Marin, G. Marteil, N. C. Fresmann, B. P. de Almeida, K. Dores, R. Fragoso, J. C. Vaz, J. B. P. Leal, J. P. Taborda Barata, S. A. Godinho, N. L. Barbosa-Morais, M. Bettencourt-Dias

This study shows centrosome clustering is common in mesenchymal tumors and leukemia, especially in cells with epithelial-to-mesenchymal transition features, and suggests targeting this process could treat certain leukemias.

bioRxiv · Mar 13, 2023 · PreprintCancer Biology

Polo-like kinase 4 (Plk4) potentiates anoikis-resistance of p53KO mammary epithelial cells by inducing a hybrid EMT phenotype

I. S. Fonseca, C. Horta, A. S. Ribeiro, B. Sousa, G. Marteil, M. Bettencourt Dias, J. Paredes

This study shows Plk4 promotes anoikis resistance in p53KO mammary epithelial cells through a hybrid EMT phenotype and P-cadherin upregulation, acting as an oncogene independent of centrosome biogenesis.

bioRxiv · Dec 16, 2022 · PreprintCancer Biology