Author

G. S. Olson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Aderem· 2 shared D. Mai· 2 shared J. Nemeth· 2 shared A. C. Rothchild· 1 shared A. Diercks· 1 shared A. H. Diercks· 1 shared A. Jahn· 1 shared A. L. Welm· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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2 shared papersImmunology

A. Aderem

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Laterally transferred macrophage mitochondria acts as a signaling source promoting cancer cell proliferation

C. U. Kidwell, J. R. Casalini, S. Pradeep, S. D. Scherer, D. Greiner, J. S. Johnson, G. S. Olson, J. Rutter, A. L. Welm, T. A. Zangle, M. Roh-Johnson

The paper shows that dysfunctional macrophage mitochondria transferred to cancer cells promote proliferation via ROS-induced ERK signaling.

bioRxiv · Aug 10, 2021Cancer Biology

Alveolar macrophages up-regulate a non-classical innate response to Mycobacterium tuberculosis infection in vivo

A. C. Rothchild, G. S. Olson, J. Nemeth, L. M. Amon, D. Mai, E. S. Gold, A. H. Diercks, A. Aderem

Alveolar macrophages infected with Mtb in vivo up-regulate an Nrf2-dependent antioxidant response instead of a pro-inflammatory response, impairing bacterial control.

bioRxiv · Mar 18, 2019 · PreprintImmunology

Latent Mycobacterium tuberculosis infection provides protection for the host by changing the activation state of the innate immune system

J. Nemeth, G. S. Olson, A. Rothchild, A. Jahn, D. Mai, F. Duffy, J. Delahaye, S. Srivatsan, C. Plumlee, K. Urdahl, E. Gold, A. Diercks, A. Aderem

This study shows that latent Mycobacterium tuberculosis infection protects hosts by altering the activation state of the innate immune system in both mice and humans.

bioRxiv · Feb 27, 2019Immunology