Author

C. U. Kidwell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

J. Rutter· 2 shared M. Roh-Johnson· 2 shared A. L. Welm· 1 shared C. Argyriou· 1 shared D. Greiner· 1 shared E. Nuebel· 1 shared G. S. Olson· 1 shared I. Wittig· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

J. Rutter

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Author profile →

2 papers

Recent·Most cited

Laterally transferred macrophage mitochondria acts as a signaling source promoting cancer cell proliferation

C. U. Kidwell, J. R. Casalini, S. Pradeep, S. D. Scherer, D. Greiner, J. S. Johnson, G. S. Olson, J. Rutter, A. L. Welm, T. A. Zangle, M. Roh-Johnson

The paper shows that dysfunctional macrophage mitochondria transferred to cancer cells promote proliferation via ROS-induced ERK signaling.

bioRxiv · Aug 10, 2021Cancer Biology

Msp1/ATAD1 restores mitochondrial function in Zellweger Spectrum Disease

E. Nuebel, J. T. Morgan, S. Fogarty, J. M. Winter, S. Lettlova, J. A. Berg, Y.-C. Chen, C. U. Kidwell, J. A. Maschek, K. J. Clowers, C. Argyriou, L. Chen, I. Wittig, J. E. Cox, M. Roh-Johnson, , J. Rutter

The paper investigates peroxisomal dysfunction in Zellweger Spectrum Disease and shows that overexpressing ATAD1 restores mitochondrial function in patient-derived fibroblasts.

bioRxiv · Sep 19, 2020 · PreprintCell Biology