Author

H. Banerjee

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. L. Szymczak-Workman· 3 shared L. P. Kane· 3 shared A. Chang· 1 shared A. Kulkarni· 1 shared B. M. Murter· 1 shared B. Murter· 1 shared C. J. Workman· 1 shared H. Nieves-Rosado· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

A. L. Szymczak-Workman

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Downregulation of PIK3IP1/TrIP on T cells is controlled by TCR signal strength, PKC, and metalloprotease-mediated cleavage

B. M. Murter, S. C. Robinson, H. Banerjee, L. Lau, U. Uche, A. L. Szymczak-Workman, L. P. Kane

This study identifies mechanisms controlling TrIP downregulation on T cells, showing it depends on TCR signal strength, PKC, and ADAM proteases.

bioRxiv · Apr 29, 2024Cell Biology

Analysis of Differential Gene Expression and Core Canonical Pathways involved in the Epithelial to Mesenchymal Transition of Triple Negative Breast Cancer Cells by Ingenuity Pathway Analysis.

H. BANERJEE, S. Bhattacharya, S. Ghosh

The paper analyzes differential gene expression and core canonical pathways in TNBC cells undergoing EMT using RNA sequencing and IPA, identifying key pathways like Sirtuin signaling and Oxidative Phosphorylation.

bioRxiv · Apr 7, 2023 · PreprintCancer Biology

Expression of Tim-3 drives naive Treg to an effector-like state with enhanced suppressive activity

H. Banerjee, H. Nieves-Rosado, A. Kulkarni, B. Murter, U. R. Chandran, A. Chang, A. L. Szymczak-Workman, L. Vujanovic, R. L. Ferris, L. P. Kane

The paper shows that Tim-3 expression in Treg cells drives them to an effector-like state with enhanced suppressive activity, promoting tumor growth.

bioRxiv · Jul 31, 2020 · PreprintImmunology

The co-stimulatory activity of Tim-3 requires Akt and MAPK signaling and immune synapse recruitment

S. Kataoka, P. Manandhar, J. Lee, C. J. Workman, H. Banerjee, A. L. Szymczak-Workman, M. Kvorjak, J. Lohmueller, L. P. Kane

This study identifies Akt and MAPK signaling as essential for Tim-3 co-stimulatory activity and shows that Tim-3 localization to the immune synapse is critical for its function.

bioRxiv · Dec 30, 2019 · PreprintImmunology