Author

H. C. Reinhardt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1

Frequent Co-Authors

M. Peifer· 2 shared A. A. Hennrich· 1 shared A. Heimsoeth· 1 shared A. Kibler· 1 shared A. Placzek· 1 shared A. Schmitt· 1 shared B. Budeus· 1 shared B. Fassbender· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCancer Biology

M. Peifer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Functional synapses between small cell lung cancer and glutamatergic neurons

A. Schmitt, V. Sakthivelu, K. Ndoci, G. A. Wani, M. Touet, I. Pintelon, I. Kisis, O. Ibruli, J. Weber, R. Maresch, C. M. Bebber, J. Goergens, M. Jevtic, F. Odenthal, A. Placzek, , F. Beleggia

This study identifies glutamatergic synaptic interactions between SCLC cells and neurons, showing that these interactions support tumor growth and can be targeted therapeutically.

bioRxiv · Jan 19, 2023Cancer Biology

A heterogeneous pool of tumor precursor cells with self-replenishing and tumor-originating properties in Chronic Lymphocytic Leukemia

B. Budeus, M. Dampmann, A. Kibler, B. Fassbender, M. Brauser, K. Bronischewski, E. Homp, S. Taudien, P. Johansson, J. Bloehdorn, S. Stilgenbauer, J. von Tresckow, J. Duerig, H. C. Reinhardt, R. Kueppers, , M. Seifert

The paper identifies a heterogeneous subpopulation of CLL-related B cells with self-replenishing and tumor-initiating properties that contribute to disease progression and clonal evolution.

bioRxiv · May 4, 2022 · PreprintCancer Biology

Loss of TP53 mediates suppression of Macrophage Effector Function via Extracellular Vesicles and PDL1 towards Resistance against Chemoimmunotherapy in B-cell malignancies

E. Izquierdo, D. Vorholt, B. Sackey, J. Nolte, S. J. Blakemore, J. Schmitz, V. Barbarino, N. Nickel, D. Bachurski, L. Lobastova, M. Nikolic, M. Michalik, R. Brinker, R. Neuhaus, L. Frenzel, , C. Pallasch

This paper identifies that TP53 loss in B-cell lymphoma leads to resistance against chemoimmunotherapy by suppressing macrophage effector function through PDL1-expressing extracellular vesicles.

bioRxiv · Jun 11, 2020 · PreprintCell Biology