Author

Hannah Hauschild

University Hospital Heidelberg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Assay:proteomics· 1

Frequent Co-Authors

Hetvi Gandhi· 1 shared Indira Dibra· 1 shared Marine BerqueziD· 1 shared Martin HelmstädteriD· 1 shared Matias SimonsiD· 1 shared Olivier DevuystiD· 1 shared Salómon Christer· 1 shared Yung-Hsin Shih· 1 shared

Activity by Year

2025-2025 · peak 1/yr

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9 top collaborators · 36 mutual co-author links

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1 shared paperCell Biology

Hetvi Gandhi

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1 paper

Recent·Most cited

A Drosophila model for Dent disease reveals impaired ER export of Cubilin as pathogenic mechanism

Salómon Christer, Zvonimir Marelja, Hannah Hauschild, Marine Berquez, Indira Dibra, Hetvi Gandhi, Yung-Hsin Shih, Martin Helmstädter, Olivier Devuyst, Matias Simons

This paper establishes a Drosophila model for Dent disease, showing that ClC-c depletion causes Cubilin ER retention and secretory pathway defects, revealing a new pathogenic mechanism.

bioRxiv · Mar 5, 2025 · PreprintCell Biology