Author

J. Byemerwa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

S. Artham· 2 shared A. D. Nguyen· 1 shared B. F. Starr· 1 shared C. D. Baeta· 1 shared C. D. Torrice· 1 shared C. Haines· 1 shared C. Joiner· 1 shared C. Lavau· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCancer Biology

S. Artham

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

UDP-6-glucose dehydrogenase in hormonally responsive breast cancers

M. J. Price, A. D. Nguyen, C. Haines, C. D. Baeta, J. Byemerwa, D. Mukherjee, S. Artham, V. Kumar, C. Lavau, S. Wardell, S. Varghese, C. R. Goodwin

This study shows that UGDH knockdown reduces estrogen-induced tumorigenic traits in breast cancer models and validates findings in human clinical specimens.

bioRxiv · Mar 20, 2024 · PreprintCancer Biology

A new chemotype of chemically tractable nonsteroidal estrogens based on a thienopyrimidine core.

V. R. Sammeta, J. D. Norris, S. Artham, C. D. Torrice, J. Byemerwa, C. Joiner, S. W. Fanning, D. P. McDonnell, T. M. Willson

The paper identifies a new chemotype of nonsteroidal estrogens based on a thienopyrimidine core through targeted screening and structure-guided optimization.

bioRxiv · Apr 15, 2022Cell Biology

Tumor genotype dictates radiosensitization after Atm deletion in brainstem gliomas

K. Deland, B. F. Starr, J. S. Mercer, J. Byemerwa, D. M. Crabtree, N. T. Williams, L. Luo, Y. Ma, M. Chen, O. J. Becher, D. G. Kirsch

This study shows that deleting Atm in p53-deficient brainstem gliomas enhances radiosensitivity, while p53 wild-type tumors respond to radiotherapy regardless of Atm status.

bioRxiv · Aug 24, 2020Cancer Biology