Author

J. D. Molkentin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 4

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:development & morphogenesis· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

A. J. York· 2 shared H. Khalil· 2 shared M. A. Sargent· 2 shared A. J. Saviola· 1 shared A. K. Johansen· 1 shared A. M. Martinson· 1 shared C. E. Murry· 1 shared D. C. Galindo· 1 shared

Activity by Year

2018-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

A. J. York

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cardiac fibroblasts regulate cardiomyocyte hypertrophy through dynamic regulation of type I collagen

Q. Meng, S. L. K. Bowers, Y. Kuwabara, J. Huo, R. Minerath, A. J. York, M. A. Sargent, V. Prasad, A. J. Saviola, D. C. Galindo, K. C. Hansen, R. J. Vagnozzi, K. E. Yutzey, J. D. Molkentin

This study shows that type I collagen produced by cardiac fibroblasts regulates cardiomyocyte hypertrophy and fibrosis, with developmental and acute deletion effects on cardiac function.

bioRxiv · May 25, 2022 · PreprintCell Biology

Inhibition of mitochondrial permeability transition by deletion of the ANT family and CypD

J. Karch, M. J. Bround, H. Khalil, M. A. Sargent, N. Latchman, N. Terada, P. M. Peixoto, J. D. Molkentin

The paper shows that genetic deletion of Ant1/2/4 and Ppif in mice fully inhibits the mitochondrial permeability transition pore.

bioRxiv · Dec 27, 2018Cell Biology

An acute immune response underlies the benefit of cardiac adult stem cell therapy

R. Vagnozzi, M. Maillet, M. Sargent, H. Khalil, A. K. Johansen, J. Schwanekamp, A. J. York, V. Huang, M. Nahrendorf, S. Sadayappan, J. D. Molkentin

This study shows that cardiac adult stem cell therapy improves heart function through an acute immune response involving macrophages, not by regenerating cardiomyocytes.

bioRxiv · Dec 26, 2018Cell Biology

Evidence for minimal cardiogenic potential of Sca-1 positive cells in the adult mouse heart

L. E. Neidig, F. Weinberger, N. J. Palpant, J. Mignone, A. M. Martinson, D. Sorensen, I. Bender, N. Nemoto, H. Reinecke, L. Pabon, J. D. Molkentin, C. E. Murry, J. van Berlo

This study investigates the cardiogenic potential of Sca-1+ cells in adult mouse hearts and finds minimal contribution to cardiomyocyte regeneration under homeostasis or after myocardial infarction.

bioRxiv · Aug 29, 2018Cell Biology