Author

J. Katz

Primary ORCID profile (0000-0002-5403-2281 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

J. KatziD1 paper · University of Alabama at Birmingham

Biology Disciplines

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Cell Biology· 1 Ecology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Research Focus:ecology & biodiversity· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. A. Khosla· 1 shared A. Chervonsky· 1 shared A. Jain· 1 shared A. Oberst· 1 shared A. S. Chawla· 1 shared B. Ghosh· 1 shared C. Jeffres· 1 shared C. Pasare· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCell Biology

A. A. Khosla

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Foodscapes for Salmon and Other Mobile Consumers in River Networks

G. Rossi, J. R. Bellmore, J. B. Armstrong, C. Jeffres, S. M. Naman, S. M. Carlson, T. E. Grantham, M. J. Kaylor, S. White, J. Katz, M. E. Power

The paper introduces the concept of 'foodscapes' to describe how food abundance, accessibility, and foraging costs vary in river networks, affecting fish growth and population stability.

bioRxiv · Aug 30, 2023Ecology

Quantifying Epithelial Plasticity as a Platform to Reverse Epithelial Injury

K. Nishida, B. Ghosh, L. Chandrala, S. Mahmud, S. Chen, A. A. Khosla, J. Katz, V. K. Sidhaye

The paper quantifies epithelial plasticity changes following injury and identifies pharmacologic targets to reverse injury in COPD.

bioRxiv · Jan 14, 2020 · PreprintCell Biology

T cells instruct dendritic cells to produce inflammasome independent IL-1β causing systemic inflammation

A. Jain, R. A. Irizarry-Caro, A. S. Chawla, N. H. Philip, K. R. Carroll, J. Katz, A. Oberst, A. Chervonsky, C. Pasare

This study identifies a T cell-instructed, inflammasome-independent pathway of IL-1β production in dendritic cells that drives systemic inflammation in autoimmune diseases.

bioRxiv · Nov 26, 2018 · PreprintImmunology