Author

J. M. Daves

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 1

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Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

A. Meyer· 1 shared B. N. Gantner· 1 shared C. M. Furdui· 1 shared D. R. Coelho· 1 shared D. R. Spitz· 1 shared F. R. Palma· 1 shared F. T. Ogata· 1 shared K. Pulakanti· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Meyer

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1 paper

Recent·Most cited

H3.1Cys96 oxidation by mitochondrial ROS promotes chromatin remodeling, breast cancer progression to metastasis and multi-drug resistance

F. R. Palma, F. T. Ogata, D. R. Coelho, K. Pulakanti, A. Meyer, Y. Huang, J. M. Daves, M. J. Schipma, C. M. Furdui, D. R. Spitz, B. N. Gantner, S. Rao, V. Backman, M. G. Bonini

The paper shows that mitochondrial H2O2 oxidizes H3.1 at Cys96, promoting chromatin remodeling, breast cancer metastasis, and drug resistance.

bioRxiv · Dec 1, 2022 · PreprintCancer Biology