Author

J. M. Kimmey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Research Focus:host-pathogen & immune response· 4 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Organism:bacteria (other)· 1 Research Focus:cell type annotation· 1 Assay:CRISPR screen· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Smirnov· 2 shared C. L. Stallings· 2 shared D. Kreamalmeyer· 2 shared R. L. Kinsella· 2 shared V. Nizet· 2 shared A. O'Donoghue· 1 shared C. LaRock· 1 shared C. Morales Fenero· 1 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersImmunology

A. Smirnov

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Author profile →

5 papers

Recent·Most cited

Streptococcus pneumoniae augments circadian clock gene expression in zebrafish cells

C. Morales Fenero, R. E. Sacksteder, J. M. Kimmey

This study shows that heat-killed Streptococcus pneumoniae increases circadian clock gene expression in zebrafish cells via a reactive oxygen species-dependent pathway.

Scientific Reports · Nov 13, 2024Peer-reviewedMicrobiology

ATG5 suppresses type I IFN-dependent neutrophil swarming and NETosis

R. L. Kinsella, C. Sur Chowdhury, A. Smirnov, Y. Mreyoud, J. M. Kimmey, E. Esaulova, D. Kreamalmeyer, M. Artyomov, C. L. Stallings

This study shows that ATG5 suppresses type I IFN-induced neutrophil swarming and NETosis during M. tuberculosis infection, contributing to disease susceptibility when ATG5 is absent.

bioRxiv · Mar 18, 2023 · PreprintImmunology

Autophagy is required to prevent early pro-inflammatory responses and neutrophil recruitment during Mycobacterium tuberculosis infection without affecting pathogen replication in macrophages

R. L. Kinsella, J. M. Kimmey, A. Smirnov, R. Woodson, M. R. Gaggioli, S. M. Chavez, D. Kreamalmeyer, C. L. Stallings

This study shows that ATG5 in CD11c+ cells suppresses pro-inflammatory cytokines and neutrophil recruitment during M. tuberculosis infection without affecting pathogen replication.

bioRxiv · Nov 4, 2022 · PreprintImmunology

Role of Inflammasome-independent Activation of IL-1β by the Pseudomonas aeruginosa Protease LasB

J. Sun, D. LaRock, E. Skowronski, J. M. Kimmey, J. Olson, Z. Jiang, A. O'Donoghue, V. Nizet, C. LaRock

This study identifies that the Pseudomonas aeruginosa protease LasB directly activates IL-1β independently of inflammasomes, contributing to lung inflammation and pathology during infection.

bioRxiv · May 18, 2020 · PreprintMicrobiology

Exploration of bacterial bottlenecks and Streptococcus pneumoniae pathogenesis by CRISPRi-seq

X. Liu, J. M. Kimmey, V. de Bakker, V. Nizet, J.-W. Veening

This study uses CRISPRi-seq to identify genetic factors important for Streptococcus pneumoniae pneumonia, revealing a critical bottleneck at 48 h post-infection and gene fitness differences between in vitro and in vivo conditions.

bioRxiv · Apr 22, 2020Microbiology