Author

J. Martin-Serrano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:electronic health records· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

W. I. Sundquist· 2 shared A. Rubio-Ramos· 1 shared A. San Paulo· 1 shared A. Yakimovich· 1 shared D. G. Miguez· 1 shared D. M. Wenzel· 1 shared D. Torrents· 1 shared I. Correas· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersCell Biology

W. I. Sundquist

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Vaccinia virus hijacks ESCRT-mediated multivesicular body formation for virus egress

M. Huttunen, A. Yakimovich, I. J. White, J. Kriston-Vizi, J. Martin-Serrano, W. I. Sundquist, J. Mercer

This study identifies ESCRT-III and VPS4 as essential for vaccinia virus packaging into multivesicular bodies, revealing an alternative pathway for virus egress.

bioRxiv · Jul 15, 2020Cell Biology

Midbody remnant inheritance is regulated by the ESCRT subunit CHMP4C

J. Casares Arias, M. U. Gonzalez, A. San Paulo, L. N. Ventimiglia, J. B. Sadler, D. G. Miguez, L. Labat-de-Hoz, A. Rubio-Ramos, L. Rangel, M. Bernabe-Rubio, J. Fernandez-Barrera, I. Correas, J. Martin-Serrano, M. A. Alonso

The paper identifies CHMP4C as a regulator of midbody remnant inheritance, showing its role in connecting MBRs to the plasma membrane and its impact on ciliation.

bioRxiv · Mar 19, 2020Cell Biology

A cancer-associated polymorphism in ESCRT-III disrupts the abscission checkpoint and promotes genome instability

J. B. A. Sadler, D. M. Wenzel, L. K. Williams, M. Guindo-Martinez, S. L. Alam, J. M. Mercader, D. Torrents, K. S. Ullman, W. I. Sundquist, J. Martin-Serrano

This paper identifies a cancer-associated polymorphism in CHMP4C that disrupts the abscission checkpoint, leading to genome instability and increased cancer risk.

bioRxiv · Jul 3, 2018Cell Biology