Author

J. P. Bolanos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:metabolism & bioenergetics· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Almeida· 2 shared J. Agulla· 2 shared M. Garcia-Macia· 2 shared R. Lapresa· 2 shared A. A. Rahim· 1 shared A. Dowsey· 1 shared A. E. Burrows· 1 shared A. J. Kowaltowski· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

A. Almeida

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Weak neuronal glycolysis sustains cognition and organismal fitness

D. Jimenez-Blasco, J. Agulla, R. Lapresa, M. Garcia-Macia, V. Bobo-Jimenez, D. Garcia-Rodriguez, I. Manjarres-Raza, E. Fernandez, Y. Jeanson, S. Khoury, J.-C. Portais, D. Padro, P. Ramos-Cabrer, P. Carmeliet, A. Almeida, J. P. Bolanos

This study shows that weak neuronal glycolysis supports cognition and organismal fitness by preventing mitochondrial redox stress and maintaining autophagy.

bioRxiv · Sep 2, 2023Cell Biology

Mitochondrial sodium/calcium exchanger NCLX regulates glycolysis in astrocytes, impacting on cognitive performance

J. V. Cabral-Costa, C. Vicente-Gutierrez, J. Agulla, R. Lapresa, J. W. Elrod, A. Almeida, J. P. Bolanos, A. J. Kowaltowski

This study shows that NCLX in astrocytes affects glycolysis and cognitive performance, with different impacts based on cell type.

bioRxiv · Sep 16, 2022Biochemistry

CLN7 mutation causes aberrant redistribution of protein isoforms and contributes to Batten disease pathobiology

A. Sharaireh, M. Guevara Ferrer, S. Herranz-Martin, M. Garcia-Macia, A. Philips, A. Tierney, M. P. Hughes, O. Coombe-Tennant, H. Nelvagel, A. E. Burrows, S. Fielding, L. M. FitzPatrick, C. D. Thornton, S. Storch, S. E. Mole, , T. R. McKay

This study shows CLN7 mutations cause nuclear and endolysosomal protein distribution defects in Batten disease, leading to apoptosis and neuroinflammation, suggesting combinatorial therapies may be needed.

bioRxiv · Apr 21, 2022 · PreprintCell Biology