Author

J. Schulte-Schrepping

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

J. L. Schultze· 3 shared C. Osei-Sarpong· 2 shared A. Al Jawazneh· 1 shared A. Condino-Neto· 1 shared A. E. Oliveira· 1 shared A. H. Marques· 1 shared A. Jaitovich· 1 shared A. Schlitzer· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersImmunology

J. L. Schultze

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Author profile →

4 papers

Recent·Most cited

BBSome-dependent ciliary Hedgehog signaling governs cell fate in the white adipose tissue

K. Sieckmann, N. Winnerling, D. J. Silva Ribeiro, R. Kardinal, L. M. Steinheuer, G. Schermann, C. Klausen, N. Blank-Stein, J. Schulte-Schrepping, C. Osei-Sarpong, M. Becker, L. Bonaguro, M. Beyer, K. Thurley, H. May-Simera, , D. Wachten

This study shows that primary cilia dysfunction in Bbs8-/- mice reduces adipose stem cells and promotes fibrogenic progenitors via disrupted Hedgehog signaling, affecting adipogenesis and lipid uptake.

bioRxiv · Jan 25, 2024 · PreprintCell Biology

Apolipoprotein E controls Dectin-1-dependent development of monocyte-derived alveolar macrophages upon pulmonary β-glucan-induced inflammatory adaptation

H. Theobald, D. Bejarano, N. Katzmarski, J. Haub, J. Schulte-Schrepping, J. Yu, K. Bassler, C. Osei-Sarpong, F. Piattini, L. Vornholz, X. Xu, S. Sheoran, A. Al Jawazneh, S. Chakarov, K. Haendler, , A. Schlitzer

This study identifies Apolipoprotein E's role in Dectin-1-dependent development of monocyte-derived alveolar macrophages during pulmonary β-glucan-induced inflammation.

bioRxiv · Sep 15, 2022Immunology

Severe COVID-19 shares a common neutrophil activation signature with other acute inflammatory states

L. F. Schimke, A. H. Marques, G. C. Baiocchi, C. A. Prado, D. L. Fonseca, P. P. Freire, D. R. Placa, I. S. Filgueira, R. C. Salgado, G. Jansen-Marques, A. E. Oliveira, J. P. S. Peron, G. Cabral Miranda, J. A. M. Barbuto, N. O. S. Camara, , O. C. Marques

This study identifies a shared neutrophil activation signature between severe COVID-19 and other acute inflammatory conditions, suggesting common therapeutic targets.

bioRxiv · Jul 30, 2021Immunology

Soluble mannose receptor induces pro-inflammatory macrophage activation and metaflammation

M. Embgenbroich, H. J. van der Zande, L. Hussaarts, J. Schulte-Schrepping, L. R. Pelgrom, N. Garcia-Tardon, L. Schlautmann, I. Stoetzel, K. Handler, J. M. Lambooij, A. Zawistowska-Deniziak, L. Hoving, K. de Ruiter, M. Wijngaarden, H. Pijl, , S. Burgdorf

This study shows that soluble mannose receptor (sMR) promotes pro-inflammatory macrophage activation and metaflammation by inhibiting CD45 phosphatase activity, leading to metabolic dysfunctions in obesity.

bioRxiv · Sep 29, 2020 · PreprintImmunology