Author

J. W. Jung

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

J. Ryu· 2 shared S. Kim· 2 shared D. Song· 1 shared D.-G. Song· 1 shared E. M. Kim· 1 shared E.-A. Shin· 1 shared G. A. Duncan· 1 shared H. Y. Kim· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

J. Ryu

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Intestinal epithelial IPMK protects mice from experimental colitis via governing colonic tuft cell development

S. E. Park, J. W. Jung, S.-H. Lee, S. J. Park, J. Ryu, S. K. Oh, S. Fang, S. Kim

This study shows that IPMK in intestinal epithelial cells protects against colitis by regulating tuft cell development and cholinergic signaling.

bioRxiv · Sep 15, 2021 · PreprintPhysiology

Rhinovirus C replication is associated with the endoplasmic reticulum and triggers cytopathic effects in an in vitro model of human airway epithelium

T. B. Gagliardi, M. E. Goldstein, D. Song, K. M. Gray, J. W. Jung, K. M. Stroka, G. A. Duncan, M. A. Scull

This study investigates RV-C15 replication in human airway epithelium, identifying the endoplasmic reticulum as the primary replication site and revealing cytopathic effects on epithelial function.

bioRxiv · Apr 16, 2021Microbiology

Differential TM4SF5-mediated SIRT1 modulation and signaling for chronic liver disease

J. Ryu, E. M. Kim, M. Kang, D.-G. Song, E.-A. Shin, J. W. Jung, S.-H. Nam, J. E. Kim, H.-J. Kim, J.-H. Lee, J.-H. Yoon, T. Son, S. Kim, H. Y. Kim, J. W. Lee

This study identifies TM4SF5's role in modulating SIRT1 and STAT3 signaling during NAFL to NASH progression, showing age-dependent effects and potential therapeutic targets.

bioRxiv · Feb 19, 2020 · PreprintCell Biology