Author

K. H. Barker

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:electrophysiology· 1

Frequent Co-Authors

D. C. Bulmer· 2 shared E. S. J. Smith· 2 shared J. P. Higham· 2 shared A. J. Brown· 1 shared F. Welsh· 1 shared I. P. Chessell· 1 shared J. Ghooraroo· 1 shared J. R. Hockley· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

D. C. Bulmer

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2 papers

Recent·Most cited

GPR35 inhibits TRPA1-mediated colonic afferent hypersensitivity through suppression of Substance P release

R. A. Gupta, J. P. Higham, P. Urriola-Munoz, K. H. Barker, L. W. Paine, J. Ghooraroo, J. R. Hockley, T. Rahman, E. S. J. Smith, A. J. Brown, R. Suzuki, D. C. Bulmer

This paper identifies GPR35 as a target for non-opioid analgesics by showing it inhibits TRPA1-mediated colonic hypersensitivity through suppression of Substance P release.

bioRxiv · Sep 12, 2023 · PreprintPhysiology

Sensitisation of colonic nociceptors by IL-13 is dependent on JAK and p38 MAPK activity

K. H. Barker, J. P. Higham, L. A. Pattison, I. P. Chessell, F. Welsh, E. S. J. Smith, D. C. Bulmer

This study shows IL-13 sensitizes colonic nociceptors via JAK and p38 MAPK signaling, offering therapeutic targets for visceral pain in gastrointestinal diseases.

bioRxiv · Oct 26, 2022 · PreprintPhysiology