Author

K. Hall

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Soldano· 1 shared A. Sparks-Wallace· 1 shared B. J. Nelson· 1 shared C. Chen· 1 shared C. Hawkins· 1 shared C. Kohne· 1 shared C. Nicksic· 1 shared J. Ghimire· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperBiochemistry

A. Soldano

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Optimization of host cell-compatible, antimicrobial peptides effective against biofilms and clinical isolates of drug-resistant bacteria.

J. Ghimire, R. J. Hart, A. Soldano, C. Chen, S. Guha, J. P. Hoffmann, K. Hall, L. Sun, B. J. Nelson, T. K. Lu, J. K. Kolls, M. Rivera, L. A. Morici, W. C. Wimley

This study optimizes antimicrobial peptides to enhance activity against drug-resistant bacteria and biofilms while reducing cytotoxicity.

bioRxiv · Dec 1, 2022Biochemistry

DNA ligase I fidelity mediates the mutagenic ligation of pol β oxidized nucleotide insertion products and base excision repair intermediates with mismatches

P. Kamble, K. Hall, M. Chandak, Q. Tang, M. Caglayan

This study shows that DNA ligase I fidelity affects the mutagenic ligation of pol β insertion products and repair intermediates with mismatches, and how APTX and FEN1 remove 5-AMP blocks.

bioRxiv · Oct 30, 2020Biochemistry

p62-mediated Selective Autophagy Endows Virus-transformed Cells with Insusceptibility to DNA Damage under Oxidative Stress

L. Wang, M. Howell, A. Sparks-Wallace, C. Hawkins, C. Nicksic, C. Kohne, K. Hall, J. Moorman, Z. Q. Yao, S. Ning

This study shows that p62-mediated selective autophagy in virus-transformed cells protects against DNA damage under oxidative stress by regulating CHK1 and RAD51 protein stability.

bioRxiv · Feb 27, 2019Cancer Biology