Author

K. K. Venneman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

D. Paine-Cabrera· 2 shared D. R. Robarts· 2 shared M. Kotulkar· 2 shared U. Apte· 2 shared C. Lau· 1 shared C. Slawson· 1 shared G. Bial· 1 shared J. A. Hanover· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPathology

D. Paine-Cabrera

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2 papers

Recent·Most cited

The Essential Role of O-GlcNAcylation in Hepatic Differentiation

D. R. Robarts, M. Kotulkar, D. Paine-Cabrera, K. K. Venneman, J. A. Hanover, N. E. Zachara, C. Slawson, U. Apte

This study shows that O-GlcNAcylation is essential for hepatocyte differentiation and its loss promotes liver fibrosis and hepatocellular carcinoma.

bioRxiv · Feb 16, 2023Pathology

Identifying Human Specific Adverse Outcome Pathways of Per- and Polyfluoroalkyl Substances Using Liver-Chimeric Humanized Mice

D. R. Robarts, D. Paine-Cabrera, M. Kotulkar, K. K. Venneman, S. Gunewardena, J. C. Corton, C. Lau, L. Foquet, G. Bial, U. Apte

This study uses liver-chimeric humanized mice to identify human-specific adverse hepatic effects of PFAS, finding NR1D1 inhibition as a common molecular mechanism.

bioRxiv · Feb 1, 2023 · PreprintPharmacology & Toxicology