Author

D. R. Robarts

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

M. Kotulkar· 3 shared U. Apte· 3 shared C. Slawson· 2 shared D. Paine-Cabrera· 2 shared J. A. Hanover· 2 shared K. K. Venneman· 2 shared N. E. Zachara· 2 shared S. Gunewardena· 2 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersPathology

M. Kotulkar

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3 papers

Recent·Most cited

The Essential Role of O-GlcNAcylation in Hepatic Differentiation

D. R. Robarts, M. Kotulkar, D. Paine-Cabrera, K. K. Venneman, J. A. Hanover, N. E. Zachara, C. Slawson, U. Apte

This study shows that O-GlcNAcylation is essential for hepatocyte differentiation and its loss promotes liver fibrosis and hepatocellular carcinoma.

bioRxiv · Feb 16, 2023Pathology

Identifying Human Specific Adverse Outcome Pathways of Per- and Polyfluoroalkyl Substances Using Liver-Chimeric Humanized Mice

D. R. Robarts, D. Paine-Cabrera, M. Kotulkar, K. K. Venneman, S. Gunewardena, J. C. Corton, C. Lau, L. Foquet, G. Bial, U. Apte

This study uses liver-chimeric humanized mice to identify human-specific adverse hepatic effects of PFAS, finding NR1D1 inhibition as a common molecular mechanism.

bioRxiv · Feb 1, 2023 · PreprintPharmacology & Toxicology

Regulation of Liver Regeneration by hepatocyte O-GlcNAcylation in mice

D. R. Robarts, S. R. McGreal, D. S. Umbaugh, W. S. Parkes, M. Kotulkar, S. Abernathy, N. Lee, H. Jaeschke, S. Gunewardena, S. A. Whelan, J. A. Hanover, N. E. Zachara, C. Slawson, U. Apte

This study shows that O-GlcNAcylation in hepatocytes is essential for proper liver regeneration, with its loss leading to uncontrolled proliferation and tumor formation.

bioRxiv · Oct 14, 2021Pathology