Author

K. T. Huang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

D. I. Yule· 2 shared A. M. Wahl· 1 shared A. Wahl· 1 shared A. Zou· 1 shared F. Zhou· 1 shared G.-Y. Son· 1 shared J. Rugis· 1 shared J. Sneyd· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersPhysiology

D. I. Yule

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2 papers

Recent·Most cited

Loss of STIM1 and STIM2 in salivary glands disrupts ANO1 function but does not induce Sjogren's disease

G.-Y. Son, A. Zou, A. Wahl, K. T. Huang, M. Vinu, S. Zorgit, F. Zhou, L. Wagner, Y. Idaghdour, D. I. Yule, S. Feske, R. S. Lacruz

This study shows that loss of STIM1 and STIM2 in salivary glands reduces ANO1 function and saliva production but does not cause Sjogren's disease, suggesting a link between SOCE and TLR8 signaling.

bioRxiv · Jan 8, 2024Physiology

The characteristics of intracellular Ca2+ signals in vivo necessitate a new model for salivary fluid secretion

D. I. Yule, T. Takano, A. M. Wahl, K. T. Huang, J. Rugis, J. Sneyd

The study observes localized Ca2+ signals in live mice that contradict previous in vitro models of salivary secretion and proposes a new mathematical model to explain the mechanism.

bioRxiv · Dec 30, 2020 · PreprintPhysiology