Author

S. Feske

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:differential expression· 2 Research Focus:cell-cell communication· 1 Assay:flow cytometry· 1

Frequent Co-Authors

F. Zhou· 2 shared G.-Y. Son· 2 shared R. S. Lacruz· 2 shared Y.-H. Wang· 2 shared A. C. Straub· 1 shared A. J. Fike· 1 shared A. L. Moreira· 1 shared A. Tao· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

F. Zhou

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Regulatory T cells and IFN-γ-producing Th1 cells play a critical role in the pathogenesis of Sjögren's Syndrome

Y.-H. Wang, W. Li, M. McDermott, G.-Y. Son, G. Maiti, F. Zhou, A. Y. Tao, D. Raphael, A. L. Moreira, B. Shen, M. Vaeth, B. Nadorp, S. Chakravarti, R. S. Lacruz, S. Feske

This study identifies Treg cells and IFN-γ-producing Th1 cells as key drivers of Sjögren's Syndrome pathogenesis using a mouse model and single-cell transcriptomics.

bioRxiv · Jan 23, 2024 · PreprintImmunology

Loss of STIM1 and STIM2 in salivary glands disrupts ANO1 function but does not induce Sjogren's disease

G.-Y. Son, A. Zou, A. Wahl, K. T. Huang, M. Vinu, S. Zorgit, F. Zhou, L. Wagner, Y. Idaghdour, D. I. Yule, S. Feske, R. S. Lacruz

This study shows that loss of STIM1 and STIM2 in salivary glands reduces ANO1 function and saliva production but does not cause Sjogren's disease, suggesting a link between SOCE and TLR8 signaling.

bioRxiv · Jan 8, 2024Physiology

Orai3 and Orai1 are essential for CRAC channel function and metabolic reprogramming in B cells

S. M. Emrich, R. E. Yoast, X. Zhang, A. J. Fike, Y.-H. Wang, K. N. Bricker, A. Tao, P. Xin, V. Walter, M. T. Johnson, T. Pathak, A. C. Straub, S. Feske, Z. Rahman, M. Trebak

This study shows Orai1 and Orai3 are essential for CRAC channel function and metabolic reprogramming in B cells, impacting proliferation, survival, and metabolic processes.

bioRxiv · May 6, 2022Immunology