Author

K.-M. Fung

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Dey· 1 shared A. S. Asch· 1 shared B. N. Bohnstedt· 1 shared C. Georgescu· 1 shared C.-H. Lee· 1 shared D. Yang· 1 shared G. Rao· 1 shared H. Homburg· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. Dey

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Activation of ERK by altered RNA splicing in cancer

Y. Zhang, M. A. Siraj, P. Chakraborty, R. Tseng, L.-T. Ku, S. Das, A. Dey, S. K. D. Dwivedi, G. Rao, M. Zhang, D. Yang, M. N. Hossen, W.-Q. Ding, K.-M. Fung, R. Bhattacharya, , P. Mukherjee

The paper shows that SMNDC1, an upregulated splicing factor in cancer, promotes tumor growth by enhancing ERK1 kinase activity through altered RNA splicing of exon 4.

bioRxiv · Aug 31, 2022 · PreprintCancer Biology

A protocol for the reduction of applied torque on parent vessel duringelastase-induced aneurysm formation using rabbit animal models

S. A. Pineda-Castillo, K. A. Laurence, H. Homburg, K.-M. Fung, B. N. Bohnstedt, C.-H. Lee

This study modifies an elastase-induced aneurysm model in rabbits to reduce parent vessel torque and improve aneurysm stability for endovascular device testing.

bioRxiv · Oct 8, 2021 · PreprintBioengineering

A Tumor Suppressor-Regulated Cell Cycle Derived Gene Signature Is Prognostic Of Recurrence Risk In Prostate Cancer

C. Georgescu, J. M. Corbin, S. Thivibilliers, Z. D. Webb, Y. D. Zhao, J. Koster, K.-M. Fung, A. S. Asch, J. D. Wren, M. J. Ruiz Echevarria

The paper develops a gene signature based on Tmeff2-regulated cell cycle genes that predicts recurrence risk in prostate cancer and outperforms existing prognostic markers.

bioRxiv · Aug 21, 2018Cancer Biology