Author

Katarina Benejova

The Ohio State University Comprehensive Cancer Center – Arthur G. James Cancer Hospital and Richard J. Solove Research Institute

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

Kristina Travis· 2 shared Martin BenejiD· 2 shared Aashrith Sai Thoutham· 1 shared Adel Fergatova· 1 shared Amy WebbiD· 1 shared Caroline DravillasiD· 1 shared Daniel J. SpakowicziD· 1 shared David Paul CarboneiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

Kristina Travis

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Targeting alpha-ketoglutarate starvation within the tumor-immune microenvironment rescues T cell effector function in cancer immunotherapy

Katarina Benejova, Kristina Travis, Deepti Mathur, No‐Joon Song, Zihai Li, Mitch A. Phelps, Elaine R. Mardis, Liza Makowski, David Paul Carbone, Dwight Hall Owen, Martin Benej

This study shows that supplementing alpha-ketoglutarate (aKG) improves T cell function and immunotherapy effectiveness in NSCLC by reversing epigenetic changes associated with T cell exhaustion.

bioRxiv · Oct 27, 2026 · PreprintCancer Biology

Mitochondrial Oxygen Consumption Drives Lung Tumor Hypoxia and Resistance to Therapy via Copy Number Alteration in Mitochondrial Electron Transport Subunit NDUFB5

Martin Benej, Katarina Benejova, Adel Fergatova, Rebecca Lisi, Kristina Travis, McKenzie Kreamer, Amy Webb, Caroline Dravillas, Rebecca Hoyd, Esin Bayralı Ülker, Aashrith Sai Thoutham, Daniel J. Spakowicz, Nicholas C. Denko

This study shows that NDUFB5 amplification in NSCLC drives tumor hypoxia and therapy resistance by increasing mitochondrial oxygen consumption.

bioRxiv · Jul 28, 2026 · PreprintCancer Biology