Author

Katherine E. Strathearn

Purdue University West Lafayette

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Aswathy ChandraniD· 1 shared Aurélie de Rus JacquetiD· 1 shared Bryce David Colon· 1 shared Daniel YsselsteiniD· 1 shared Deniz KirikiD· 1 shared Gad G. YousefiD· 1 shared George P. McCabeiD· 1 shared J. C. Rochet· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperNeuroscience

Aswathy ChandranORCID

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2 papers

Recent·Most cited

Protective effects of polyphenol-rich extracts against neurotoxicity elicited by paraquat or rotenone in cellular models of Parkinson's disease

Mitali Arun Tambe, Aurélie de Rus Jacquet, Katherine E. Strathearn, Jennifer A. Hensel, Bryce David Colon, Aswathy Chandran, Gad G. Yousef, Mary H. Grace, Mário G. Ferruzzi, Qingli Wu, James Edward Simon, Mary Ann Lila, Jean‐Christophe Rochet

The paper investigates the neuroprotective effects of polyphenol-rich extracts against paraquat and rotenone-induced neurotoxicity in Parkinson's disease models, identifying mechanisms involving antioxidant pathways and mitochondrial function.

Antioxidants · Jul 20, 2023Peer-reviewedNeuroscience

Two C-terminal sequence variations determine differential neurotoxicity between human and mouse α-synuclein

Natalie Landeck, Katherine E. Strathearn, Daniel Ysselstein, Kerstin Buck, Sayan Dutta, Siddhartha Banerjee, Zhengjian Lv, John D. Hulleman, Jagadish Hindupur, Li‐Kai Lin, Sonal Padalkar, George P. McCabe, Lia Stanciu, Yuri L. Lyubchenko, Deniz Kirik, J. C. Rochet

The paper compares neurotoxicity of human A53T and mouse α-synuclein variants, finding mouse aSyn is less toxic due to C-terminal substitutions reducing membrane-induced aggregation and vesicle permeabilization.

Molecular Neurodegeneration · Sep 8, 2020Peer-reviewedNeuroscience