Author

Katherine Papastratos

The University of Melbourne

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Ebtihal H MustafaiD· 2 shared Julia V. MilneiD· 2 shared Kameng Wu· 2 shared Maree Pechlivanis· 2 shared Nicholas James ClemonsiD· 2 shared Niko ThioiD· 2 shared Sasha WittsiD· 2 shared Wayne Allen PhillipsiD· 2 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

Ebtihal H MustafaORCID

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2 papers

Recent·Most cited

Mapping Functional Tumor Suppressor Networks in Esophageal Adenocarcinoma Using In Vivo CRISPR Screening and Perturb-sequencing

Ebtihal H Mustafa, Katherine Papastratos, Kameng Wu, Niko Thio, Julia V. Milne, Natasha Mitchell, Sasha Witts, Maree Pechlivanis, Bonnie Wong, Nicola Waddell, Wayne Allen Phillips, Nicholas James Clemons

The paper identifies tumor suppressors and their functional roles in esophageal adenocarcinoma progression using CRISPR screening and Perturb-seq.

bioRxiv · Jul 21, 2026 · PreprintCancer Biology

SMAD4 loss drives chromosomal instability during tumourigenesis via translational reprogramming

Julia V. Milne, Kameng Wu, Eric P. Kusnadi, Sandra Brosda, Kenji Mark Fujihara, Ebtihal H Mustafa, Sasha Witts, Katherine Papastratos, Maree Pechlivanis, Anna Trigos, Metta K. Jana, Paul J. McMillan, Thomas Daniel Jackson, Niko Thio, Yuzhou Feng, , Nicholas James Clemons

This study shows SMAD4 loss reprograms translation to induce chromosomal instability, driving tumourigenesis, and identifies CDK11B-p58 as a key mediator.

bioRxiv · Mar 25, 2026 · PreprintCancer Biology