L. Dolat

The acetylase activity of Cdu1 regulates bacterial exit from infected cells by protecting Chlamydia effectors from degradation

The study shows that Cdu1's acetylase activity protects Chlamydia effectors from degradation, enabling bacterial exit from host cells.

Chlamydia trachomatis effector Dre1 interacts with dynactin to reposition host organelles during infection

This study identifies Dre1, a Chlamydia effector that interacts with dynactin to reposition host organelles during infection, enhancing bacterial fitness.

An endometrial organoid model of Chlamydia-epithelial and immune cell interactions

This paper develops an endometrial organoid model to study Chlamydia trachomatis infection and its effects on host epithelial cells and immune cell recruitment.

The effector TepP mediates the recruitment and activation of Phosphoinositide 3 Kinase on early Chlamydia trachomatis vacuoles

This study identifies TepP as a Chlamydia trachomatis effector that recruits and activates PI3K on early vacuoles, modulating host signaling and gene expression.