Author

L. Dumoutier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Diefenbach· 1 shared A. Ohnemus· 1 shared A. Papageorgiou· 1 shared A. Wack· 1 shared C. A. Reis· 1 shared C. Blanchetot· 1 shared C. Michiels· 1 shared D. Schnepf· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. Diefenbach

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2 papers

Recent·Most cited

Structural basis for the mechanism and antagonism of receptor signaling mediated by Interleukin-9 (IL-9)

T. De Vos, M. Godar, F. Bick, A. Papageorgiou, T. Evangelidis, I. Markovic, E. Mortier, L. Dumoutier, K. Tripsianes, C. Blanchetot, S. N. Savvides

This study reveals the structural basis of IL-9 signaling and antagonism through crystallography and NMR, showing how IL-9 binds its receptor and how antibodies block its activity.

bioRxiv · Dec 30, 2022 · PreprintImmunology

Rotavirus susceptibility of antibiotic-treated mice ascribed to diminished expression of interleukin-22

D. Schnepf, P. Hernandez, T. Mahlakoiv, S. Crotta, M. E. Sullender, S. T. Peterson, A. Ohnemus, C. Michiels, I. Gentle, L. Dumoutier, C. A. Reis, A. Diefenbach, A. Wack, M. T. Baldridge, P. Staeheli

This study shows that antibiotic-treated mice are more susceptible to rotavirus due to reduced IL-22 expression, which normally inhibits viral replication through mechanisms independent of IFN signaling.

bioRxiv · Feb 18, 2021Immunology