Author

S. T. Peterson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Organism:mouse· 4 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:development & morphogenesis· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

C.-A. Assenmacher· 2 shared D. Sorobetea· 2 shared I. E. Brodsky· 2 shared J. P. Grayczyk· 2 shared M. Lanza· 2 shared M. T. Baldridge· 2 shared R. Matsuda· 2 shared A. C. Bohrer· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersImmunology

C.-A. Assenmacher

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

A TNF-IL-1 circuit controls Yersinia within intestinal granulomas

R. Matsuda, D. Sorobetea, J. Zhang, S. T. Peterson, J. P. Grayczyk, B. Herrmann, W. Yost, R. O'Neill, A. C. Bohrer, M. Lanza, C.-A. Assenmacher, K. D. Mayer-Barber, S. Shin, I. E. Brodsky

This study identifies a monocyte-intrinsic TNF-IL-1 circuit that controls Yersinia infection within intestinal granulomas by signaling to non-hematopoietic cells.

bioRxiv · Apr 21, 2023 · PreprintImmunology

Inflammatory monocytes promote pyogranuloma formation to counteract Yersinia blockade of host defense

D. Sorobetea, R. Matsuda, S. T. Peterson, J. P. Grayczyk, I. Rao, E. Krespan, M. Lanza, C.-A. Assenmacher, D. P. Beiting, E. Radaelli, I. E. Brodsky

This study shows that Yersinia pseudotuberculosis induces pyogranulomas in the murine intestine by blocking phagocytosis, and that monocytes are essential for granuloma formation and immune control.

bioRxiv · Feb 6, 2022 · PreprintMicrobiology

A homeostatic interferon-lambda response to bacterial microbiota stimulates preemptive antiviral defense within discrete pockets of intestinal epithelium

J. A. Van Winkle, S. T. Peterson, E. A. Kennedy, M. J. Wheadon, H. Ingle, C. Desai, R. Rodgers, D. A. Constant, A. P. Wright, L. Li, M. Artyomov, S. Lee, M. T. Baldridge, T. J. Nice

The paper shows bacterial microbiota induce localized IFN-lambda-driven antiviral defenses in intestinal epithelial cells, enhancing host resistance to enteric viruses.

bioRxiv · Jun 2, 2021Immunology

Rotavirus susceptibility of antibiotic-treated mice ascribed to diminished expression of interleukin-22

D. Schnepf, P. Hernandez, T. Mahlakoiv, S. Crotta, M. E. Sullender, S. T. Peterson, A. Ohnemus, C. Michiels, I. Gentle, L. Dumoutier, C. A. Reis, A. Diefenbach, A. Wack, M. T. Baldridge, P. Staeheli

This study shows that antibiotic-treated mice are more susceptible to rotavirus due to reduced IL-22 expression, which normally inhibits viral replication through mechanisms independent of IFN signaling.

bioRxiv · Feb 18, 2021Immunology