Author

L. G. Williams

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

A. H. Meijer· 1 shared A. Lewis· 1 shared F. R. Hammond· 1 shared G. F. Wiegertjes· 1 shared H. E. Anderson· 1 shared J. D. Strauss· 1 shared J. G. Jackson· 1 shared K. P. Hodel· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. H. Meijer

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

An arginase 2 promoter transgenic illuminates anti-inflammatory signalling in zebrafish

F. R. Hammond, A. Lewis, H. E. Anderson, L. G. Williams, A. H. Meijer, G. F. Wiegertjes, P. M. Elks

This study develops a zebrafish transgenic model to investigate anti-inflammatory signaling, showing arginase 2 is expressed in neutrophils during immune challenges.

bioRxiv · Feb 14, 2022 · PreprintImmunology

Mouse model and human patient data suggest critical roles for Pten and p53 in suppressing POLE mutant tumor development

V. S. Park, M. J. Sun, W. D. Frey, L. G. Williams, K. P. Hodel, J. D. Strauss, S. J. Wellens, J. G. Jackson, Z. F. Pursell

The paper shows that Pten and p53 loss contribute to POLE mutant tumor growth, but not mutation burden, in mice and humans.

bioRxiv · Oct 10, 2021 · PreprintCancer Biology