Author

L. Mendez

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Evolutionary Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

B. Ibanez· 1 shared C. D. Barratt· 1 shared C. Escudero· 1 shared D. Caamano· 1 shared E. A. Escudero-Guevara· 1 shared E. Sebastian· 1 shared E. Vuelta· 1 shared F. O. Castro· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperPhysiology

B. Ibanez

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Extracellular vesicles from preeclampsia disrupt the blood-brain barrier via reduced claudin-5: potential role of vascular endothelial growth factor

H. Sandoval, B. Ibanez, M. Contreras, F. Troncoso, F. O. Castro, D. Caamano, L. Mendez, E. A. Escudero-Guevara, H. D. Mistry, L. O. Kurlak, M. Vatish, J. Acurio, C. Escudero

This study shows that extracellular vesicles from preeclampsia and hypoxic placentae reduce claudin-5 in the blood-brain barrier, potentially via VEGF, leading to BBB disruption and neurological deficits.

bioRxiv · Apr 10, 2024 · PreprintPhysiology

Genomic signatures of past megafrugivore-mediated dispersal in Malagasy palms

L. Mendez, C. D. Barratt, W. Durka, W. D. Kissling, W. L. Eiserhardt, W. J. Baker, V. Randrianasolo, R. E. Onstein

The paper examines how past megafrugivore extinctions in Madagascar affected genetic diversity and differentiation in Malagasy palms, finding higher diversity in megafruit species and links between extinct dispersers and reduced genetic differentiation.

bioRxiv · Jan 20, 2023Evolutionary Biology

CRISPR/Cas9 technology abolishes the BCR/ABL1 oncogene effect in chronic myeloid leukemia and restores normal hematopoiesis

E. Vuelta, J. L. Ordonez, V. Alonso-Perez, L. Mendez, P. Hernandez-Carabias, R. Saldana, J. Sevilla, E. Sebastian, S. Muntion, F. Sanchez-Guijo, J. M. Hernandez-Rivas, I. Garcia-Tunon, M. A. Sanchez-Martin

This study uses CRISPR/Cas9 to target and disrupt the BCR/ABL1 oncogene in CML cells, restoring normal hematopoiesis in mouse models and patient-derived xenografts.

bioRxiv · Aug 5, 2020 · PreprintCancer Biology