Author

Lorena Decker

Universität Ulm

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Axel FreischmidtiD· 2 shared Sonja MengeiD· 2 shared Benedikt BrorsiD· 1 shared Dávid BrenneriD· 1 shared Jochen Hans WeishauptiD· 1 shared Johannes C. M. SchlachetzkiiD· 1 shared Karin ForsbergiD· 1 shared Karin M. DanzeriD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCell Biology

Axel FreischmidtORCID

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2 papers

Recent·Most cited

Loss of FXR1 and FXR2 promotes accumulation of TDP-43 in aging-related stress granules

Sonja Menge, Lorena Decker, Karin M. Danzer, Patrick Oeckl, Axel Freischmidt

The paper shows that loss of FXR1 and FXR2 leads to TDP-43 accumulation in stress granules, linking FXP family reduction to neurodegenerative pathology.

bioRxiv · Oct 27, 2025 · PreprintCell Biology

Somatic mutations in ALS genes in the motor cortex of sporadic ALS patients

Óscar González-Velasco, Rosanna Parlato, Rüstem Yilmaz, Lorena Decker, Sonja Menge, Axel Freischmidt, Xiaoxu Yang, Nikshitha Tulasi, Dávid Brenner, Peter Munch Andersen, Karin Forsberg, Johannes C. M. Schlachetzki, Benedikt Brors, Lena Voith von Voithenberg, Jochen Hans Weishaupt

This study identifies somatic mutations in ALS genes in the motor cortex of sporadic ALS patients, particularly in excitatory neurons, suggesting a role in disease initiation.

bioRxiv · Mar 31, 2025 · PreprintGenetics