Author

M. A. Pufall

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

C. A. Musselman· 1 shared C. M. Rush· 1 shared C. R. Hagan· 1 shared C.-H. Yang· 1 shared D. H. Lum· 1 shared J. A. O. Zimmerman· 1 shared J. C. Sanchez· 1 shared J. Gertz· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperBiochemistry

C. A. Musselman

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3 papers

Recent·Most cited

PI3Kδ inhibition potentiates glucocorticoids in B-ALL by decreased receptor phosphorylation and enhanced gene regulation

J. A. O. Zimmerman, M. Fang, M. A. Pufall

This study shows that inhibiting PI3Kδ enhances glucocorticoid efficacy in B-ALL by reducing GR phosphorylation and improving gene regulation.

bioRxiv · Feb 10, 2023 · PreprintCancer Biology

Characterization of a novel estrogen- and progesterone-responsive endometrial cancer cell line: HCI-EC-23

C. M. Rush, Z. Blanchard, J. T. Polaski, K. S. Osborne, K. Osby, J. M. Vahrenkamp, C.-H. Yang, D. H. Lum, C. R. Hagan, K. K. Leslie, M. A. Pufall, K. W. Thiel, J. Gertz

This paper introduces a new endometrial cancer cell line, HCI-EC-23, which responds to estrogen and progesterone, and shows it has similar estrogen-responsive gene regulation to Ishikawa cells.

bioRxiv · Aug 25, 2022Cancer Biology

The Molecular Basis of Specific DNA Binding by the BRG1 AT-hook and Bromodomain

J. C. Sanchez, L. Zhang, S. Evoli, N. J. Schnicker, M. Nunez-Hernandez, L. Yu, J. Wereszczynski, M. A. Pufall, C. A. Musselman

This study identifies the DNA specificity and structural basis of BRG1's composite domain using SELEX-seq, NMR, and modeling, showing how cancer mutations alter DNA binding.

bioRxiv · Nov 25, 2019Biochemistry