Author

M. C. Jarvis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Cristini· 1 shared A. Z. Cheng· 1 shared B. Stefanovska· 1 shared C. Attarian· 1 shared C. Beghe· 1 shared C. J. Bierle· 1 shared D. Salamango· 1 shared E. Law· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Cristini

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

R-loop homeostasis and cancer mutagenesis promoted by the DNA cytosine deaminase APOBEC3B

J. McCann, A. Cristini, E. Law, S. LEE, M. Tellier, M. Carpenter, C. Beghe, J. Kim, M. C. Jarvis, B. Stefanovska, N. A. Temiz, E. N. Bergstrom, D. Salamango, M. Brown, S. Murphy, , R. Harris

This study shows APOBEC3B regulates R-loop homeostasis and contributes to mutagenesis in cancer by binding R-loops and altering their landscape.

bioRxiv · Aug 30, 2021 · PreprintCancer Biology

Drying of virus-containing aerosol particles: Modelling effects of droplet origin and composition.

This paper models how virus-containing aerosol droplets dry, showing differences in solute concentration based on droplet origin and composition, with implications for virus survival.

bioRxiv · Mar 25, 2021Biophysics

A Conserved Mechanism of APOBEC3 Relocalization by Herpesviral Ribonucleotide Reductase Large Subunits

A. Z. Cheng, S. Nobrega de Moraes, C. Attarian, J. Yockteng-Melgar, M. C. Jarvis, M. Biolatti, G. Galitska, V. Dell'Oste, L. Frappier, C. J. Bierle, S. A. Rice, R. S. Harris

This study shows that herpesviruses use viral ribonucleotide reductase subunits to relocalize APOBEC3B and APOBEC3A, suggesting a conserved mechanism to evade innate immunity.

bioRxiv · Sep 13, 2019Microbiology