Author

M. Ethier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:rat· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

G. Fergusson· 3 shared J. Ghislain· 3 shared V. Poitout· 3 shared C. Tremblay· 2 shared H. Maachi· 2 shared A. Guillaume· 1 shared A.-L. Castell· 1 shared C. Goubault· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersPhysiology

G. Fergusson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Beta-cell mass expansion during puberty involves serotonin signaling and determines glucose homeostasis in adulthood

A.-L. Castell, M. Ethier, G. Fergusson, C. Tremblay, C. Goubault, M. Baltz, D. Dal Soglio, J. Ghislain, V. Poitout

This study shows that puberty increases beta-cell proliferation via GH/serotonin signaling, and metabolic stress during puberty affects glucose homeostasis in adulthood.

bioRxiv · Apr 6, 2022Physiology

Combined deletion of free fatty-acid receptors 1 and 4 minimally impacts glucose homeostasis in mice

M. L. Croze, A. Guillaume, M. Ethier, G. Fergusson, C. Tremblay, S. A. Campbell, H. Maachi, J. Ghislain, V. Poitout

This study shows that deleting both FFAR1 and FFAR4 in mice only slightly affects glucose homeostasis, with minor changes in fasting glucose and glucose tolerance.

bioRxiv · Aug 4, 2020Physiology

HB-EGF Signaling is Required for Glucose-Induced Pancreatic β-Cell Proliferation in Rats

H. Maachi, G. Fergusson, M. Ethier, G. N. Brill, L. S. Katz, L. B. Honig, M. R. Metukuri, D. K. Scott, J. Ghislain, V. Poitout

This study shows HB-EGF signaling is essential for glucose-induced β-cell proliferation in rats, mediated through EGFR and ChREBP pathways.

bioRxiv · Dec 3, 2019Cell Biology