Author

L. S. Katz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:mouse· 1 Assay:proteomics· 1 Organism:rat· 1

Frequent Co-Authors

D. K. Scott· 3 shared A. Garcia-Ocana· 1 shared C. Jean-Pierre· 1 shared C. Ottmann· 1 shared E. J. Visser· 1 shared G. Brill· 1 shared G. Fergusson· 1 shared G. N. Brill· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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3 shared papersCell Biology

D. K. Scott

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Molecular glues of the regulatory ChREBP/14-3-3 complex protect beta cells from glucolipotoxicity.

L. S. Katz, E. J. Visser, K. F. Plitzko, M. Pennings, P. J. Cossar, I. L. Tse, M. Kaiser, L. Brunsveld, C. Ottmann, D. K. Scott

This paper identifies molecular glues that stabilize the ChREBP/14-3-3 complex, protecting beta cells from glucolipotoxicity by preventing ChREBP-mediated dedifferentiation.

bioRxiv · Feb 16, 2024Cell Biology

Nrf2 Regulates β-cell Mass by Suppressing Cell Death and Promoting Proliferation

S. Baumel-Alterzon, L. S. Katz, G. Brill, C. Jean-Pierre, Y. Li, S. Biswal, A. Garcia-Ocana, D. K. Scott

This study shows that Nrf2 regulates β-cell mass by reducing cell death and enhancing proliferation, suggesting it as a therapeutic target for diabetes.

bioRxiv · Mar 5, 2021 · PreprintPhysiology

HB-EGF Signaling is Required for Glucose-Induced Pancreatic β-Cell Proliferation in Rats

H. Maachi, G. Fergusson, M. Ethier, G. N. Brill, L. S. Katz, L. B. Honig, M. R. Metukuri, D. K. Scott, J. Ghislain, V. Poitout

This study shows HB-EGF signaling is essential for glucose-induced β-cell proliferation in rats, mediated through EGFR and ChREBP pathways.

bioRxiv · Dec 3, 2019Cell Biology