Author

M. Gan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 3 Assay:DNA methylation· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:bacteria (other)· 1 Research Focus:host-pathogen & immune response· 1 Approach:machine learning (classical)· 1 Approach:mechanistic / dynamical model· 1

Frequent Co-Authors

B. Li· 1 shared D. Geng· 1 shared F. Xiao· 1 shared G. Ge· 1 shared G. Hu· 1 shared G. Verkhivker· 1 shared G.-P. Zhao· 1 shared H. Fu· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCell Biology

B. Li

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Mycobacterial DnaQ is an Alternative Proofreader Ensuring DNA Replication Fidelity

M.-Z. Deng, Q. Liu, S.-J. Cui, H. Fu, M. Gan, Y.-Y. Xu, X. Cai, W. Sha, G.-P. Zhao, S. M. Fortune, L.-D. Lyu

This study shows that mycobacterial DnaQ acts as an alternative proofreader, working with the PHP domain to maintain DNA replication fidelity and prevent mutations.

bioRxiv · Oct 24, 2023 · PreprintMicrobiology

GLI1 facilitates rheumatoid arthritis by collaborative regulation of DNA methyltransferases

G. Ge, Q. Guo, Y. Zhou, W. Li, W. Zhang, J. Bai, Q. Wang, H. Tao, W. Wang, Z. Wang, M. Gan, Y. Xu, H. Yang, B. Li, D. Geng

This study shows GLI1 promotes rheumatoid arthritis by regulating DNA methyltransferases, leading to M1 macrophage polarization and osteoclast activation, and suggests GLI1 inhibitors as potential treatments.

bioRxiv · Feb 7, 2023Cell Biology

Dissecting Mutational Allosteric Effects in Alkaline Phosphatases Associated with Different Hypophosphatasia Phenotypes: An Integrative Computational Investigation

F. Xiao, Z. Zhou, X. Song, M. Gan, J. Long, G. Verkhivker, G. Hu

This paper uses computational methods to analyze ALPL mutations in Hypophosphatasia, finding that severe mutations significantly affect allosteric communications and protein stability.

bioRxiv · Jan 24, 2022Biophysics