Author

M. Gaspari

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Alaimo· 1 shared A. Antoccia· 1 shared A. Bähr· 1 shared A. Bertossi· 1 shared A. Bisio· 1 shared A. Dendorfer· 1 shared A. Filippi· 1 shared A. Lunardi· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCancer Biology

A. Alaimo

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

TRAP1 and cyclophilin D compete at OSCP subunit to regulate enzymatic activity and permeability transition pore opening by F-ATP synthase

G. Cannino, A. Urbani, M. Gaspari, M. Varano, A. Negro, A. Filippi, F. Ciscato, I. Masgras, C. Gerle, E. Tibaldi, A. M. Brunati, G. Lippe, P. Bernardi, A. Rasola

bioRxiv · Dec 6, 2021Cell Biology

Intra-epithelial non-canonical Activin A signalling safeguards prostate progenitor quiescence

F. Cambuli, V. Foletto, A. Alaimo, D. De Felice, F. Gandolfi, M. D. Palumbieri, M. Zaffagni, S. Genovesi, M. Lorenzoni, M. Celotti, E. Bertossio, G. Mazzero, A. Bertossi, A. Bisio, F. Berardinelli, , A. Lunardi

This study identifies non-canonical Activin A signaling as a mechanism maintaining prostate epithelial progenitor quiescence, with implications for cancer and therapy.

bioRxiv · Mar 5, 2021 · PreprintCancer Biology

Primate heart regeneration via migration and fibroblast repulsion by human heart progenitors

C. Schneider, K. S. Foo, M. T. De Angelis, G. Santamaria, F. Reiter, T. Dorn, A. Bähr, Y. L. Tsoi, P. Hoppmann, I. My, A. Meier, V. Jurisch, N. Hornaschewitz, S. Schwarz, K. Lu, , K.-L. Laugwitz

This study shows human heart progenitors can regenerate primate hearts by migrating to injury sites, repelling fibroblasts, and replacing damaged tissue through CXCL12/CXCR4 and SLIT2/ROBO1 signaling.

bioRxiv · Jul 3, 2020 · PreprintCell Biology