Author

M. Krasilnikov

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:fluorescence / live-cell microscopy· 1

Frequent Co-Authors

T. Peng· 2 shared A. A. Chang· 1 shared A. B. Molofsky· 1 shared A. L. Gan· 1 shared A. Molofsky· 1 shared A. N. Mattis· 1 shared A. Shen· 1 shared C. Feliciano· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersImmunology

T. Peng

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Group 2 innate lymphoid cells constrain type 3/17 lymphocytes in shared stromal niches to restrict liver fibrosis

J. Sbierski-Kind, K. M. Cautivo, J. Wagner, M. W. Dahlgren, J. Nilsson, M. Krasilnikov, N. M. Mroz, C. O. Lizama, A. L. Gan, P. R. Matatia, M. T. Taruselli, A. A. Chang, S. Caryotakis, C. O'Leary, M. Kotas, , A. B. Molofsky

The paper investigates the role of IL-5+ lymphocytes and type 3/17 lymphocytes in liver fibrosis, showing their spatial relationship and cross-regulation in fibroblast niches.

bioRxiv · Apr 26, 2023 · PreprintImmunology

Development of a dual fluorescent reporter system in Clostridioides difficile reveals a division of labor between virulence and transmission gene expression

M. L. Donnelly, S. D. Shrestha, J. W. Ribis, P. Kuhn, C. Feliciano, A. Shen, M. Krasilnikov

This study develops a dual fluorescent reporter system to analyze toxin and sporulation gene expression in C. difficile, revealing a division of labor between virulence and transmission gene expression under specific growth conditions.

bioRxiv · Mar 3, 2022Microbiology

Sentinel p16INK4a+ cells in the basement membrane form a reparative niche in the lung

N. Reyes de Mochel, K. N. Cheong, M. Cassandras, C. Wang, M. Krasilnikov, P. Matatia, A. Molofsky, J. Campisi, T. PENG

The paper identifies p16INK4a+ mesenchymal cells in the lung basement membrane as reparative sentinels that transition to a senescence-associated secretory phenotype to support epithelial repair.

bioRxiv · Jun 10, 2020 · PreprintCell Biology