M. Landim-Vieira

This study identifies sMyBP-C's critical role in skeletal muscle development and function using gene-edited mouse models, showing its necessity for prenatal and postnatal muscle growth and structural integrity.

This study identifies NFκB signaling in cardiac myocytes as a driver of myocardial injury and arrhythmias in a mouse model of arrhythmogenic cardiomyopathy.

The paper identifies PTMs on β-MHC in human hearts and shows their changes in failing hearts, suggesting acetylation levels are influenced by PTM location rather than disease type.

This paper introduces a method to quantify myofibrillar orientation in cardiac muscle and shows it detects greater disorientation in diseased tissue compared to healthy controls.