Author

M. Marquet

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1

Frequent Co-Authors

C. Brandt· 2 shared A. Garot· 1 shared A. Henke· 1 shared A. S. Mosig· 1 shared A. Viehweger· 1 shared B. Löffler· 1 shared B. Vuong· 1 shared C. Carrion· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersMicrobiology

C. Brandt

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The IgH Eμ-MAR regions promote UNG-dependent error-prone repair to optimize somatic hypermutation

O. Martin, M. Thomas, M. Marquet, A. Garot, M. Brousse, S. Bender, C. Carrion, J. E. Choi, B. Vuong, P. Gearhart, R. Maul, S. Le Noir, E. Pinaud

The paper shows that IgH Eμ-MAR regions limit error-prone repair to the variable region of Ig genes by promoting UNG-dependent repair, affecting somatic hypermutation patterns.

bioRxiv · Aug 15, 2022 · PreprintImmunology

Nanopore-based enrichment of antimicrobial resistance genes -- a case study

A. Viehweger, M. Marquet, M. Hölzer, N. Dietze, M. Pletz, C. Brandt

This study evaluates adaptive nanopore sequencing for detecting antimicrobial resistance genes in clinical isolates, showing its effectiveness compared to standard methods.

bioRxiv · Aug 29, 2021Microbiology

SARS-CoV-2 causes severe alveolar inflammation and barrier dysfunction

S. Deinhardt-Emmer, S. Böttcher, C. Häring, L. Giebeler, A. Henke, R. Zell, F. Hornung, C. Brandt, M. Marquet, A. S. Mosig, M. W. Pletz, M. Schacke, J. Roedel, R. Heller, S. Nietzsche, , C. Ehrhardt

This study shows SARS-CoV-2 infects alveolar epithelial cells, causing inflammation and barrier dysfunction, while endothelial cells are damaged through cytokine release from infected epithelial cells.

bioRxiv · Aug 31, 2020Microbiology